Chronic Fatigue Syndrome Alternative Treatments Ebook
Chronic fatigue immune dysfunction syndrome (CFIDS) first came to the public's attention in the mid-1980s. It primarily strikes young people between the ages of 25 and 40, with women under 45 accounting for 80 percent of the cases. CFIDS is characterized by overwhelming fatigue and other flulike symptoms. It is not contagious and does not result from overexertion. Symptoms can be sudden and debilitating, but are not fatal. There is no known cause. Theories of a connection between CFIDS and Epstein-Barr virus (which causes mononucleosis) are apparently unfounded.
Patients with the chronic fatigue syndrome (CFS) often come to the attention of specialists in rehabilitation. A lot of sociopolitical baggage accompanies this diagnosis. Consumer groups and clinicians who believe in specific causes may villify those who do not. The prevalence of at least 6 months of new-onset fatigue unexplained by a medical diagnosis is 0.5 in American women and 0.3 in men.192 These persons report a lower functional status than matched patients with congestive heart fail-ure.193 The syndrome may include fibro-myalgia, depression, and an irritable bowel. Al
Most pediatric patients who require inpatient care are hospitalized in a general or specialty pedi-atric unit under the care of a pediatrician, or with joint care also provided by child and adolescent mental health services. Patients with diagnoses of conversion disorder or severe chronic fatigue syndrome may also be hospitalized in adolescent psychiatry units. Some centers have developed adolescent medical wards, which have proven very helpful for adolescents with complex combined medical and psychiatric needs. One academic unit, directed by Professor Elena Garralda at Imperial College, London, has a particular interest in psychosomatic medicine.
Diagnosing fibromyalgia is difficult because we don't know what causes this condition. Patients are often frustrated because they have met many physicians who say, It's all in your head. There have been links to the onset of symptoms and exposure to Epstein-Barr virus (EBV) that causes mononucleosis. Since pain and fatigue are key symptoms, others have thought it may be connected to chronic fatigue syndrome (CFS).
What happens if you don't replenish your muscle-glycogen stores Simple If you run out of glycogen, you run out of energy. The amount of muscle fuel you have determines how long you can exercise. As a car needs a full tank of gas before heading out on a long trip, an endurance athlete requires sufficient muscle gasoline to sustain the pace and go the distance. Always tired or run down Obviously, a vigorous training schedule alone is enough to make you feel that way. You might also want to look into your carbohydrate consumption. Keep a food log and do the math there could be an easy solution to your problem.
A different explanation of incubation effects is the recovery from fatigue theory. This rather straightforward idea states that when people are fatigued, their tired minds are not up to the task of solving a difficult problem. After a break, they are mentally refreshed, and better able to solve difficult problems. This clear explanation has some support clearly refreshed minds will be better able than tired ones to solve difficult problems. On the other hand, many experiments have controlled for mental fatigue, and have shown that neither fatigue, nor relief from fatigue are necessary for incubation effects to occur.
It is clearly true that incubation effects are multiply caused. That means that there are many causes of incubation effects, and furthermore, that different cases of incubation effects occur for different reasons. Scientific studies show clearly that incubation periods can allow initial fixation to be forgotten, or escaped. Other experimental studies show that when people are frustrated in their preliminary attempts to solve problems, they remain sensitive to stimuli related to the problem, and serendipitously encountered hints can trigger incubation effects. Furthermore, recovering from mental fatigue after a rest can cause incubation, and conscious work that is forgotten may appear to cause incubation effects. Finally, combinations of these factors are also likely the multiple causes of incubation effects that people experience.
Tics that persist through later childhood and into adolescence have a characteristic natural history. Follow-up studies indicate that tics on average increase gradually through the grade school years before peaking at age 11 (Leckman et al., 1998). Tics then gradually decline in average severity through adolescence until stabilizing at relatively low levels by young adulthood. Superimposed on this gradual rise, plateau, and decline in average tic severity is a fluctuation, or waxing and waning, of baseline tic severity. Although this fluctuation is nonrandom (Peterson and Leckman, 1998), the biological determinants of the fluctuations remain poorly characterized. Clinical experience has repeatedly shown that emotional stress, physical fatigue, and excitement can reliably exacerbate tics. Neuroendocrine studies provide supportive evidence for an important role of stress in modulating the severity of tic symptoms (Chappell et al., 1994).
Titrated to maintain a serum testosterone concentration of 20 nM (Howell et al. 2001). Testosterone treatment did not alter bone turnover markers, hip, spine or forearm bone mineral density (quantitative computed tomography and DEXA), lean mass or fat mass (DEXA), mood (hospital anxiety and depression scale) or sexual function. However, two out of five components of the multi-dimensional fatigue inventory were improved (activity was increased and physical fatigue was reduced). These inconsistent and minor effects were supported by a case control study showing minimal differences based on lower serum testosterone concentrations in similar men (Howell et al. 2000) suggesting that androgen replacement therapy offers little objective benefit for men with compensated Leydig cell failure post-cytotoxic therapy.
Oriental Medicine is particularly well suited to look at your chronic fatigue. Work with your acu-pro to figure out a treatment strategy that realistically meets your expectations and goals. I'm a provider for the Fibromyalgia CFIDS Network, which connects members with qualified practitioners who understand and frequently treat those conditions. Your CFIDS symptoms may be similar to others with the same diagnosis, but your overall health will determine the Oriental Medical pattern that needs attention and the treatment plan that will be customized for you. Your acu-pro, in considering the patterns below, also includes the CFIDS symptoms listed earlier in this chapter. Common CFIDS patterns include Your CFIDS symptoms can improve greatly and be well maintained with Oriental Medicine. The Fibromyalgia CFIDS Network provides members with the latest recommended treatments and provider lists for this condition. Acupuncture is frequently mentioned as a viable treatment option. Check...
Focus was more effective than a treatment-as-usual condition in improving depression and global functioning in youth with inflammatory bowel disease and mild depression. Not only did overall depressive severity improve, but neurovegetative symptoms (e.g., fatigue, appetite changes, sleep changes) usually attributed to the inflammatory disease process itself also improved significantly immediately after treatment (Szigethy et al. 2009). In another study of 66 adolescents with chronic fatigue syndrome, participants randomized to CBT had significantly decreased fatigue, improved physical functioning, and better school attendance over a longer period of 2 years compared with a wait-list control group (Knoop et al. 2008). Interestingly, rating of severity of maternal fatigue was a significant predictor of treatment outcome.
Barlow and Ellard (2004) reviewed 12 psycho-educational intervention studies, including samples of children with asthma, chronic fatigue syndrome, diabetes, juvenile arthritis, and chronic pain. Overall, studies showed evidence of effectiveness on such variables such as self-efficacy, self-management of disease, family functioning, psychosocial well-being, reduced isolation, social competence, knowledge,
DSPS that presents in adolescence is sometimes misdiagnosed as narcolepsy, depression, chronic fatigue, or as a learning disorder. Knowledge that sleepiness occurs mainly in the morning and continuous restful sleep occurs when patients go to bed late and arise late helps the clinician arrive at the correct diagnosis.
Immune systems, 151 nausea, 149-150 pain, 150-151 carpal tunnel syndrome, 75-77 CFIDS, 221 childbirth, 160-162 constipation, 188-189 coughing, 126 cupping, 43 dental pain, 62-64 depression, 208-209 dianhea, 190 151-153 chemotherapy, 149-150 herbal medicines, 153-154 immune systems, 151 pain, 150-151 CFIDS, 221 childbirth, 160-162 choosing, 243-244 constipation, 187-189 cystitis, 181-184 dental pain, 62-64 depression, 208-209 diagnosis Chinese Medicine, 278 Traditional Chinese Medical College of Hawaii, 278 carpal tunnel syndrome, 75-76 CFIDS, 221
The predominant subjective symptom during OT seems to be the feeling of 'heavy legs', rather than muscle soreness, not only during unusually low exercise intensities but also during daily routine activities. Furthermore athletes frequently complain about chronic fatigue and sleep disturbances.
Growth hormone (GH somatotropin) is secreted by the anterior pituitary in response to hypothalamic GH-releasing hormone (GHRH). GH promotes linear growth and has both anabolic and catabolic effects. Hypersecretion of GH causes gigantism when it occurs before epiphyseal closure and acromegaly when it begins afterward. Hyposecretion causes short stature in childhood and possibly a chronic fatigue-like syndrome in adults. GH acts indirectly through insulin-like growth factors (IGFs, somatomedins), with IGF-1 (formerly somatomedin C) being the most important for growth. Prolonged exposure to elevated levels of GH and IGF-1 results in the insidious onset of skeletal and soft tissue overgrowth, the latter of which is most pronounced in tissues containing large amounts of cartilage proteoglycans. 109 Cortical bone density is increased and trabecular bone (e.g., vertebral) density is decreased, probably because of coexistent hypogonadism. W Soft tissue hypertrophy may result in compression...
Other diagnostic considerations in patients who complain of long periods of unrefreshing sleep include atypical depression and chronic fatigue syndrome. In both of these disorders, patients often spend many hours in bed, but actual sleep time is increased only mildly or not at all. Idiopathic hypersomnia is usually considered in a patient who complains of sleepiness and has no apparent cause for sleepiness based on the clinical history and a nocturnal polysomnogram. For such patients, the differential diagnosis includes narcolepsy, upper airway resistance syndrome, insufficient sleep syndrome, and obstructive sleep apnea that occurs during some nights but not others. The diagnosis of idiopathic hypersomnia is not easy because it requires ruling out all other causes to the extent possible.
Research in the United Kingdom is funded by a number of funding organizations, including the Department of Health, the Wellcome Trust, and the Medical Research Council. Additional research is funded by special-interest charities dedicated to such diseases as diabetes mellitus or Crohn's disease. Major topics of research interest include recognition of mental health problems by family doctors and pediatricians, chronic fatigue, and the epidemiology of pediatric mental health.
This is particularly true for somatization disorder, which occurs in 10 -20 of first-degree relatives of patients with this disorder. Somatization disorders show a concordance rate of 29 in monozygotic twin studies (Kaplan et al. 1994). Rates of anxiety and depression are also higher in family members of somatiz-ing children and adolescents, suggesting a possible genetic etiology (Fritz et al. 1997). Mothers of children who somatize are more likely to report an excess of functional abdominal pain, anxiety, depression, and other somatic symptoms and are more likely to have a history of irritable bowel syndrome, chronic fatigue, and somatoform disorder (Campo et al. 2007). A study by Marshall et al. (2007) suggests that compared with children of parents with long-term medical illness, children of parents with somatoform disorders tend to demonstrate higher levels of problematic health cognitions, as do their parents. Some studies indicate that parental...
Pain are most common among prepubertal children, whereas complaints of limb pain, fatigue, and muscle aches appear to increase in frequency with age. Specific constellations of symptoms may result in the diagnosis of syndromes such as irritable bowel syndrome, chronic fatigue, and or fibromyalgia. Patients with somatization disorder often have a history of concurrent treatment from several physicians, which typically results in fragmented care and contradictory treatment plans, along with multiple workups for the same symptoms. Comorbid anxiety, depressive, conduct, and or substance abuse symptoms are common.
The key feature of depression is a negative view of oneself, of the world, and of the future. There is loss of interest and energy and slowing of mental function. Mental symptoms include a sense of loss, sadness, hopelessness, and pessimism about the future. There may be disturbances of appetite, sleep, and sexual function. Physical symptoms such as headache, constipation, weakness, aches, and pain are also common. Simon et al (1999) found that about 50 of patients with major depression have multiple unexplained bodily symptoms. Many of them present with somatic symptoms but acknowledge psychological symptoms when asked about them. Eleven percent deny any psychological symptoms, even on direct questioning. In patients with chronic low back pain, the most common depressive symptoms include sleep disturbance, loss of energy, chronic fatigue, and persistent worrisome thoughts (Rush et al 2000). (See Main & Spanswick 2000 p 203 for the ICD-10 and DSM-IV diagnostic...
Arsenic encephalitis, continuous progressive headaches, physical and mental fatigue, vertigo, restlessness, mild somnolence, and focal paresis develop. Spinal cord involvement leads to weakness, sphincter disturbances, motor and sensory impairment, and trophic changes. Optic neuritis manifested by cloudy vision and visual field defects may also be observed subacutely but can be delayed for as long as 2 years. Generally, a mixed sensory and motor neuropathy develops within 7 to 10 days after ingestion of toxic amounts of arsenic, and patients often complain of severe burning in the soles of the feet (Fig. 39-2 (Figure Not Available) ). Long-standing cognitive changes have been reported.
Few investigators have examined whether youth with JRA experience cognitive sequelae that are associated with treatment or disease variables (Carter et al. 1999 Feldmann et al. 2005). There has been some evidence to suggest that the use of steroids associated with the management of JRA may produce cognitive sequelae (Feldmann et al. 2005). Employing a case-control design, Carter et al. (1999) examined cognitive functioning in youth with JRA and youth with chronic fatigue. Cognitive functioning was measured with the Kaufman Brief Intelligence Test (K-BIT). Findings revealed no significant differences between the two groups, with both groups having a mean K-BIT intelligence quotient that was in the average range. This suggested that cognitive functioning of youth with JRA actually may not be impaired.
Patients with chronic severe MR often complain of gradual and progressive dyspnea on exertion. They may also experience chronic fatigue and weakness due to a low cardiac output. On exam, patients with MR often demonstrate a harsh, pansystolic murmur, predominantly at the apex, but frequently radiating to the axilla or neck. A systolic click may be present in patients with mitral valve prolapse. A thrill may occasionally be palpated at the apex. The murmur should not change with inspiration, but may increase with Valsalva maneuver. The apical impulse may be hyperkinetic. Lung exam is frequently normal in chronic MR, while the chest roentgenogram shows cardiomegaly and left atrial enlargement. The electrocardiogram often demonstrates left ventricular hypertrophy, left atrial enlargement, and frequently the rhythm is atrial fibrillation.
The notion of impaired cortical drive as a mechanism of fatigue is compatible with the fMRI findings shown in Figure 3-7 for a patient with demonstrable fatigability with exercise. Demyelination of the U-fibers under the primary motor cortex representation for the back and hip presumably limited sensory drive and output from this region. Functional MRI may reveal other correlates with fatigue, such as hypoactivity in corticocortical and cortico-subcortical networks for motor planning and the execution of movements. Other investigators found an association between symptoms of fatigue and a reduction in glucose metabolism by 18F-fluorodeoxyglucose PET in the frontal cortex and the basal ganglia caused by subcortical demyelination.116 Impaired motor planning or other cognitive functions associated with diminished prefrontal drive may also contribute to fatigue. An abnormal balance between cortical excitation and inhibition, based on changes in EEG frequencies during simple motor tasks,...
V Find relief from chronic fatigue syndrome using acu-points and herbs Do you have your Qi running pedal to the metal Does your energy lurch forward like a rocketing drag racer with high blood pressure, or does your chronic fatigue engine cough and sputter as you crawl out of the starting gate In this chapter, you'll discover the help that Oriental Medicine has been giving to all kinds of Qi drivers, including those who stay in one lane or those who weave across all three lanes because of an addition to harmful substances.
Clinical Features and Associated Findings. Infectious mononucleosis is the only known disease caused by acute EBV infection. The neurological complications of infectious mononucleosis include meningoencephalitis, Guillain-Barre syndrome, cerebellitis, transverse myelitis, cranial nerve palsies of which the most common is a seventh nerve palsy, and a variety of optic nerve abnormalities (papilledema, optic neuritis, retrobulbar neuritis). 'W EBV infection is almost universally associated with primary central nervous system lymphomas in patients with AIDS.'wi Persistent viral activity or reactivation of EBV has been studied in the etiology of chronic fatigue syndrome. Chronic Fatigue Syndrome. Chronic fatigue syndrome is characterized by debilitating fatigue accompanied by a variety of symptoms including cognitive difficulty, The diagnosis of primary CNS lymphoma is based on CT or MRI evidence of a focal enhancing mass lesion or lesions, and typically an unsuccessful response to...
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