Conclusion

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Defining inflammatory status in patients with ACS and in those with coronary atherosclerosis is becoming paramount to the efficient risk assessment and appropriate treatment of patients. As our grasp of molecular and genetic tools improves, the ability to break down the complex pathways and modulate them as potential therapeutic targets in susceptible individuals will take hold. The implications are far reaching and hold the potential to significantly alter the way clinical medicine is practiced. Nowhere is this more relevant than in interventional cardiology. From novel devices and instruments in PCI to adjuvant therapy focused on modulating inflammation, clinical practice will evolve over time. Whether this translates into improved clinical outcomes is yet to be determined. However, what will take place is a more individualized strategy of therapy based on a more complex profile of risk, one that incorporates inflammation as a vital component.

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