Genetics

Multiple genetic polymorphisms have been associated with hypertension. It is estimated that up to 30% to 50% of variability in BP may have a genetic basis.11 The majority of these polymorphisms appear to be involved directly or indirectly in renal sodium reabsorption, which may represent future therapeutic drug targets. 2 In addition to the identification of genetic factors contributing to the development of hypertension, explorations into the genetic basis of variability in response to drug therapy are at early stages. Although advances are made on both fronts, replication of early findings

identifying genetic variations from genome-wide association scans (GWAS) clearly require more comprehensive study before investigators, let alone clinicians, are in a position to utilize these findings.1

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