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fluorescence studies strengthen the conclusion that differences in midpiece volumes in human beings, and other mammals, do spring from functional differences in mitochondrial loading and sperm bioenergetics.

Two further points may be made as regards sperm midpiece volumes in mammals. Firstly, with rare exceptions, the mitochondria in the sperm midpiece do not participate in development of the fertilized egg. They are excluded, and it is only the mitochondria contained in the ovum, and deriving from the female parent, which contribute to embryonic development. This being the case, it might be argued that advances in mitochondrial efficiency

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